Allopathy.

Beginning of the new decade was highlighted with the global outbreak of Coronavirus disease-19 (COVID-19). It is a kind of viral pneumonia which is caused by severe acute respiratory syndrome coronavirus-2 (SARS-CoV-2), a new strain of Coronavirus. This outbreak of pneumonia with unknown etiology was thought to have originated from the Huanan seafood market and was reported firstly at Wuhan, in China. On 31st January 2020, the World Health Organization (WHO) announced that COVID-19 was listed as the Public Health Emergency of International Concern (PHEIC).The virus was named as COVID-19 on 11th February, 2020 by WHO. As it has affected the whole world, it was declared as global pandemic by WHO. SARS-CoV-2 is the betacoronavirus with presence of different proteins which helps in attachment, penetration, biosynthesis, maturation and release of newly synthesized virion particles in the host body and infect the host. COVID-19 is contagious and primarily spreads through the respiratory tract, by droplets, respiratory secretions, and direct contact. As no vaccine or drug molecule are available yet, breaking the chain, enhancing an individual's body immunity, identifying the infection early and timely medical care are the best ways to prevent COVID-19 infection. Along with Allopathy medicines, Ayurveda, Unani and Homeopathy medicines are used as add-ons to treat the disease. Thus, taking into consideration this global threat this review focuses on origin, morphology of SARS-CoV-2, pathogenesis, role of Ayurveda, Unani, Homeopathy and Allopathy in treatment of COVID-19 and the current scenario of vaccines against COVID-19.

The concept of hyperuricemia has not been described at all in Unani system of medicine, but description of a joint disorder known as Niqris, has been found in most of the classical Unani texts. The clinical features of Niqris very much resemble with the Gout as described in Allopathy, which occurs due to hyperuricemia. In practice, hyperuricemia is frequently defined as a serum urate levels exceeding of 7mg/dl (0.42 mmol/l) in adult males and 6 mg/dl (0.36 mmol/l) in adult females. However, this arbitrary definition of Hyperuricemia has been justified by epidemiological studies showing that those with SUA levels greater than 7mg/dl are at increased risk of developing gouty arthritis and urolithiasis. Hyperuricemia is fairly common, with a prevalence ranging between 2.3 to 41.4 percent in various populations. A variety of factors appear to be associated with higher serum urate concenterations. The present study was conducted on 50 patients of Primary Hyperuricemia attending the OPD of Ajmal Khan Tibbiya College and Hospital. During the study it was observed that the mean serum uric acid level, which was 8.6±1.1before the beginning of the study got reduced to 6.6±1.3 at the end of the study. As we applied paired “t” test to the observations recorded before and after treatment it was found that t=12.6, p